The problem of genital endometriosis is of special importance nowadays due to a steady increase in its prevalence among gynecological patients (10% in recent years) (1, 2). The disease mainly affects women in their reproductive age (90%-99% of patients are aged 20-50 years), regardless of their ethnicity or socio-economic status (2). Despite the thorough attention to this problem worldwide, many aspects of etiology and pathogenesis of this disease remain controversial (3, 4). Genetic predisposition, retrograde menstruation, coelomic metaplasia, activation of embryonic remnants, changes in the metabolism of endometrial cells, increased proliferative activity and autonomous growth of endometrioid heterotopias, and iatrogenic causes are considered to be the main etiological factors of endometriosis. Risk factors for the development of endometriosis include hyperestrogenemia,

The problem of genital endometriosis is of special importance nowadays due to a steady increase in its prevalence among gynecological patients (10% in recent years) (1, 2). The disease mainly affects women in their reproductive age (90%-99% of patients are aged 20-50 years), regardless of their ethnicity or socio-economic status (2). Despite the thorough attention to this problem worldwide, many aspects of etiology and pathogenesis of this disease remain controversial (3, 4). Genetic predisposition, retrograde menstruation, coelomic metaplasia, activation of embryonic remnants, changes in the metabolism of endometrial cells, increased proliferative activity and autonomous growth of endometrioid heterotopias, and iatrogenic causes are considered to be the main etiological factors of endometriosis. Risk factors for the development of endometriosis include hyperestrogenemia,